MAPK4 overexpression promotes tumor progression via noncanonical activation of AKT/mTOR signaling-Reference-Cited by-同舟云学术

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MAPK4 overexpression promotes tumor progression via noncanonical activation of AKT/mTOR signaling

Published:2019-01-28 Issue:3 Volume:129 Page:1015-1029
ISSN:0021-9738
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:

Author:

Wang Wei^ORCID,Shen Tao,Dong Bingning,Creighton Chad J.,Meng Yanling,Zhou Wolong,Shi Qing,Zhou Hao,Zhang Yinjie,Moore David D.^ORCID,Yang Feng^ORCID

Funder

Department of Defense Congressionally Directed Medical Research Programs

Cancer Prevention Research Institute of Texas

Adrienne Helis Malvin Medical Research Foundation

National Institute of Health

Publisher

American Society for Clinical Investigation

Subject

General Medicine

Link

https://www.jci.org/articles/view/97712/files/pdf

Reference35 articles.

1. Association of phosphatidylinositol kinase activity with polyoma middle-T competent for transformation

2. PDGF-dependent tyrosine phosphorylation stimulates production of novel polyphosphoinositides in intact cells

3. The protein kinase encoded by the Akt proto-oncogene is a target of the PDGF-activated phosphatidylinositol 3-kinase

4. Characterization of a 3-phosphoinositide-dependent protein kinase which phosphorylates and activates protein kinase Bα

5. mTOR: from growth signal integration to cancer, diabetes and ageing

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