A perspective on anti-CCN2 therapy for chronic kidney disease
Author:
Publisher
Oxford University Press (OUP)
Subject
Transplantation,Nephrology
Link
http://academic.oup.com/ndt/article-pdf/29/suppl_1/i30/6909331/gft430.pdf
Reference68 articles.
1. The origin of interstitial myofibroblasts in chronic kidney disease
2. Origin and function of myofibroblasts in kidney fibrosis
3. The origin of scar-forming kidney myofibroblasts
4. CCN proteins: multifunctional signalling regulators
5. Connective tissue growth factor coordinates chondrogenesis and angiogenesis during skeletal development
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1. CCN2 Activates RIPK3, NLRP3 Inflammasome, and NRF2/Oxidative Pathways Linked to Kidney Inflammation;Antioxidants;2023-07-31
2. Long noncoding RNA AI662270 promotes kidney fibrosis through enhancing METTL3‐mediated m6A modification of CTGF mRNA;The FASEB Journal;2023-06-30
3. Discovery and Design of Novel Cyclic Peptides as Specific Inhibitors Targeting CCN2 and Disrupting CCN2/EGFR Interaction for Kidney Fibrosis Treatment;Journal of Medicinal Chemistry;2023-06-06
4. Cellular communication network 2 (connective tissue growth factor) aggravates acute DNA damage and subsequent DNA damage response-senescence-fibrosis following kidney ischemia reperfusion injury;Kidney International;2022-12
5. Interference with connective tissue growth factor attenuated fibroblast-to-myofibroblast transition and pulmonary fibrosis;Annals of Translational Medicine;2022-05
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