Stability and low induction propensity of cefiderocol against chromosomal AmpC β-lactamases of Pseudomonas aeruginosa and Enterobacter cloacae
Author:
Affiliation:
1. Shionogi & Co., Ltd, Toyonaka, Osaka, Japan
Publisher
Oxford University Press (OUP)
Subject
Infectious Diseases,Pharmacology (medical),Pharmacology,Microbiology (medical)
Link
http://academic.oup.com/jac/article-pdf/73/11/3049/26144743/dky317.pdf
Reference17 articles.
1. AmpC β-lactamases;Jacoby;Clin Microbiol Rev,2009
2. Interplay of efflux system, ampC, and oprD expression in carbapenem resistance of Pseudomonas aeruginosa clinical isolates;Quale;Antimicrob Agents Chemother,2010
3. Altered outer membrane transcriptome balance with AmpC overexpression in carbapenem-resistant Enterobacter cloacae;Majewski;Front Microbiol,2016
4. Risk factors and clinical features of infections caused by plasmid-mediated AmpC β-lactamase-producing Enterobacteriaceae;Park;Int J Antimicrob Agents,2009
5. In vitro activity of the siderophore cephalosporin, cefiderocol, against a recent collection of clinically relevant Gram-negative bacilli from North America and Europe, including carbapenem-nonsusceptible isolates (SIDERO-WT-2014 study);Hackel;Antimicrob Agents Chemother,2017
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