IL‐17A promotes lung fibrosis through impairing mitochondrial homeostasis in type II alveolar epithelial cells

Author:

Xiao Huijuan12ORCID,Peng Liang3,Jiang Dingyuan2,Liu Yuan24,Zhu Lili2,Li Zhen2,Geng Jing2,Xie Bingbing2,Huang Xiaoxi5,Wang Jing6,Dai Huaping2,Wang Chen12

Affiliation:

1. Department of Pulmonary and Critical Care Medicine, Center of Respiratory Medicine, China‐Japan Friendship Hospital, School of Clinical Medicine Peking University Beijing China

2. Department of Pulmonary and Critical Care Medicine, Center of Respiratory Medicine, China‐Japan Friendship Hospital; National Center for Respiratory Medicine; National Clinical Research Center for Respiratory Diseases, Institute of Respiratory Medicine, Chinese Academy of Medical Sciences Peking Union Medical College Beijing China

3. Beijing Key Laboratory for Immune‐Mediated Inflammatory Diseases, Institute of Medical Science China‐Japan Friendship Hospital Beijing China

4. Department of Respiratory and Critical Care Medicine Zhongnan Hospital of Wuhan University Wuhan China

5. Medical Research Center Beijing Chaoyang Hospital Affiliated to Capital Medical University Beijing China

6. State Key Laboratory of Medical Molecular Biology, Department of Physiology, Institute of Basic Medical Sciences Chinese Academy of Medical Sciences School of Basic Medicine Peking Union Medical College Beijing China

Funder

National Natural Science Foundation of China

National Key Research and Development Program of China

Publisher

Wiley

Subject

Cell Biology,Molecular Medicine

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