Sulforaphane improves disrupted ER-mitochondria interactions and suppresses exaggerated hepatic glucose production-Reference-Cited by-同舟云学术

Sulforaphane improves disrupted ER-mitochondria interactions and suppresses exaggerated hepatic glucose production

Published:2018-02 Issue: Volume:461 Page:205-214
ISSN:0303-7207
Container-title:Molecular and Cellular Endocrinology
language:en
Short-container-title:Molecular and Cellular Endocrinology

Author:

Tubbs Emily^ORCID,Axelsson Annika S.^ORCID,Vial Guillaume,Wollheim Claes B.,Rieusset Jennifer,Rosengren Anders H.

Funder

Crafoord foundation

Swedish Foundation for Strategic Research

Ragnar Söderberg foundation

Publisher

Elsevier BV

Subject

Endocrinology,Molecular Biology,Biochemistry

Reference40 articles.

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4. Mitochondrial dysfunction results from oxidative stress in the skeletal muscle of diet-induced insulin-resistant mice;Bonnard;J. Clin. Invest.,2008

5. Identification of PTEN at the ER and MAMs and its regulation of Ca(2+) signaling and apoptosis in a protein phosphatase-dependent manner;Bononi;Cell Death Differ.,2013

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