TNIP2 mediates GRβ-promoted inflammation and is associated with severity of major depressive disorder-Reference-Cited by-同舟云学术

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TNIP2 mediates GRβ-promoted inflammation and is associated with severity of major depressive disorder

Published:2021-07 Issue: Volume:95 Page:454-461
ISSN:0889-1591
Container-title:Brain, Behavior, and Immunity
language:en
Short-container-title:Brain, Behavior, and Immunity

Author:

Chiang Ting-I,Hung Yi-Yung,Wu Ming-Kung,Huang Ya-Ling,Kang Hong-Yo

Funder

Ministry of Science and Technology, Taiwan

Kaohsiung Chang Gung Memorial Hospital

National Science Council

Publisher

Elsevier BV

Subject

Behavioral Neuroscience,Endocrine and Autonomic Systems,Immunology

Reference57 articles.

1. Homodimerization of the glucocorticoid receptor is not essential for response element binding: activation of the phenylethanolamine N-methyltransferase gene by dimerization-defective mutants;Adams;Mol. Endocrinol.,2003

2. Differential expression of glucocorticoid receptor transcripts in major depressive disorder is not epigenetically programmed;Alt;Psychoneuroendocrinology,2010

3. Context-dependent cooperation between nuclear factor kappaB (NF-kappaB) and the glucocorticoid receptor at a TNFAIP3 intronic enhancer: a mechanism to maintain negative feedback control of inflammation;Altonsy;J. Biol. Chem.,2014

4. TNIP2 is a Hub Protein in the NF-kappaB Network with Both Protein and RNA Mediated Interactions;Banks;Mol. Cellular Proteomics : MCP,2016

5. Inflammatory activation is associated with a reduced glucocorticoid receptor alpha/beta expression ratio in monocytes of inpatients with melancholic major depressive disorder;Carvalho;Transl. Psychiatry,2014

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