Friedreich Ataxia: current state-of-the-art, and future prospects for mitochondrial-focused therapies-Reference-Cited by-同舟云学术

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Friedreich Ataxia: current state-of-the-art, and future prospects for mitochondrial-focused therapies

Published:2021-03 Issue: Volume:229 Page:135-141
ISSN:1931-5244
Container-title:Translational Research
language:en
Short-container-title:Translational Research

Author:

Pallardó Federico V.,Pagano Giovanni,Rodríguez Laura R.,Gonzalez-Cabo Pilar,Lyakhovich Alex,Trifuoggi Marco

Funder

Universitat de València

Publisher

Elsevier BV

Subject

Biochemistry (medical),Public Health, Environmental and Occupational Health,General Medicine,Physiology (medical)

Reference60 articles.

1. Frataxin is reduced in Friedreich ataxia patients and is associated with mitochondrial membranes;Campuzano;Hum Mol Genet,1997

2. Mitochondrial dysfunction in neurons in Friedreich's ataxia;Stepanova;Mol Cell Neurosci,2020

3. Antioxidant therapies and oxidative stress in Friedreich's ataxia: the right path or just a diversion?;Rodríguez;Antioxidants,2020

4. Friedreich's ataxia: clinical features, pathogenesis and management;Cook;Br Med Bull,2017

5. Clinical features of Friedreich's ataxia: classical and atypical phenotypes;Parkinson;J Neurochem,2013

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