Calcium entry via TRPC6 mediates albumin overload-induced endoplasmic reticulum stress and apoptosis in podocytes-Reference-Cited by-同舟云学术

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Calcium entry via TRPC6 mediates albumin overload-induced endoplasmic reticulum stress and apoptosis in podocytes

Published:2011-12 Issue:6 Volume:50 Page:523-529
ISSN:0143-4160
Container-title:Cell Calcium
language:en
Short-container-title:Cell Calcium

Author:

Chen Shan,He Fang-Fang,Wang Hui,Fang Zhan,Shao Ning,Tian Xiu-Juan,Liu Jian-She,Zhu Zhong-Hua,Wang Yu-Mei,Wang Sheng,Huang Kai,Zhang Chun

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology,Physiology

Reference29 articles.

1. Hereditary proteinuria syndromes and mechanisms of proteinuria;Tryggvason;N. Engl. J. Med.,2006

2. The podocyte's response to injury: role in proteinuria and glomerulosclerosis;Shankland;Kidney Int.,2006

3. Transforming growth factor-beta1 is up-regulated by podocytes in response to excess intraglomerular passage of proteins: a central pathway in progressive glomerulosclerosis;Abbate;Am. J. Pathol.,2002

4. Glomerular podocyte vacuolation in focal segmental glomerulosclerosis;Yoshikawa;Arch. Pathol. Lab. Med.,1986

5. Podocyte injury induced by albumin overload in vivo and in vitro: involvement of TGF-beta and p38 MAPK;Yoshida;Nephron. Exp. Nephrol.,2008

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2. New Frontiers on ER Stress Modulation: Are TRP Channels the Leading Actors?;International Journal of Molecular Sciences;2022-12-22

3. TRPC6 Inactivation Reduces Albuminuria Induced by Protein Overload in Sprague Dawley Rats;Cells;2022-06-21

4. Hypercalciuria switches Ca2+ signaling in proximal tubular cells, induces oxidative damage to promote calcium nephrolithiasis;Genes & Diseases;2022-03

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