Autophagy impairment with lysosomal and mitochondrial dysfunction is an important characteristic of oxidative stress-induced senescence-Reference-Cited by-全球学者库

Autophagy impairment with lysosomal and mitochondrial dysfunction is an important characteristic of oxidative stress-induced senescence

Published:2016-11-21 Issue:1 Volume:13 Page:99-113
ISSN:1554-8627
Container-title:Autophagy
language:en
Short-container-title:Autophagy

Author:

Tai Haoran¹,Wang Zhe¹,Gong Hui¹,Han Xiaojuan¹,Zhou Jiao¹,Wang Xiaobo¹,Wei Xiawei¹,Ding Yi¹,Huang Ning¹,Qin Jianqiong¹,Zhang Jie¹,Wang Shuang¹,Gao Fei²,Chrzanowska-Lightowlers Zofia M.²,Xiang Rong³,Xiao Hengyi¹

Affiliation:

1. Lab for Aging Research, Center of Gerontology and Geriatrics, State Key Laboratory of Biotherapy & Collaborative Innovation Center of Biotherapy, West China Hospital, Sichuan University, Chengdu, China

2. Wellcome Trust Center for Mitochondrial Research, Institute of Neuroscience, Newcastle University, Newcastle upon Tyne, UK

3. Department of Clinical Medicine, Medical School of Nankai University, Tianjin, China

Publisher

Informa UK Limited

Subject

Cell Biology,Molecular Biology

Link

https://www.tandfonline.com/doi/pdf/10.1080/15548627.2016.1247143

Reference48 articles.

1. Cellular and molecular mechanisms of stress-induced premature senescence (SIPS) of human diploid fibroblasts and melanocytes

2. Senescence-like growth arrest induced by hydrogen peroxide in human diploid fibroblast F65 cells.

3. A biomarker that identifies senescent human cells in culture and in aging skin in vivo.