Cellular and molecular mechanisms of stress-induced premature senescence (SIPS) of human diploid fibroblasts and melanocytes

Author:

Toussaint O,Medrano E.E,von Zglinicki T

Publisher

Elsevier BV

Subject

Cell Biology,Endocrinology,Genetics,Molecular Biology,Ageing,Biochemistry

Reference106 articles.

1. Involvement of the cyclin-dependent kinase inhibitor p16 (INK4a) in replicative senescence of normal human fibroblasts;Alcorta;Proc. Natl. Acad. Sci. USA,1996

2. UVB induces atypical melanocytic lesions and melanoma in human skin;Atillasoy;Am. J. Pathol.,1998

3. Oxygen modulates growth of human cells at physiologic partial pressures;Balin;J. Exp. Med.,1984

4. Melanin accumulation accelerates melanocyte senescence by a mechanism involving p16INK4a/CDK4/pRB and E2F1;Bandyopadhyay;Ann. N.Y. Acad. Sci.,2000

5. Comparison of the responses of human melanocytes with different melanin contents to ultraviolet B irradiation;Barker;Cancer Res.,1995

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