1 Loss of IP3 Receptor-Mediated Ca2+ Release in Mouse B Cells Results in Abnormal B Cell Development a..
来源:J IMMUNOL( P 0022-1767 E 1550-6606 ) 发表时间: 2017/07
类型:期刊论文 为本人加分:1329.072544
2 Heat Shock Protein 60 in Cardiovascular Physiology and Diseases
来源:FRONT MOL BIOSCI( P E 2296-889X ) 发表时间: 2020/04
类型:期刊论文 为本人加分:272.812367
贡献度:共同第一作者
3 SETD2 is essential for terminal differentiation of erythroblasts during fetal erythropoiesis
来源:BIOCHEM BIOPH RES CO( P 0006-291X E 1090-2104 ) 发表时间: 2021/05
类型:期刊论文 为本人加分:195.931914
贡献度:共同第一作者;共同通讯作者
4 IP3R-mediated Ca2+ signaling controls B cell proliferation through metabolic reprogramming
来源:ISCIENCE( P E 2589-0042 ) 发表时间: 2022/05
类型:期刊论文 为本人加分:117.556064
贡献度:共同第一作者;共同通讯作者
5 MAMPs: A devil tamed becomes an angel
来源:CELL HOST MICROBE( P 1931-3128 E 1934-6069 ) 发表时间: 2023/09
类型:期刊论文 为本人加分:41.457616
6 Loss of IP3R-dependent Ca2+ signalling in thymocytes leads to aberrant development and acute lymphob..
来源:NAT COMMUN( P 2041-1723 E ) 发表时间: 2014/09
类型:期刊论文 为本人加分:29.242356
7 Deletion of heat shock protein 60 in adult mouse cardiomyocytes perturbs mitochondrial protein homeo..
来源:CELL DEATH DIFFER( P 1350-9047 E 1476-5403 ) 发表时间: 2020/02
类型:期刊论文 为本人加分:20.730134
贡献度:参与作者
8 IP3R-mediated Ca2+ signals govern hematopoietic and cardiac divergence of Flk1(+) cells via the calc..
来源:J MOL CELL BIOL( P 1674-2788 E 1759-4685 ) 发表时间: 2017/08
类型:期刊论文 为本人加分:14.348335
贡献度:参与作者
9 Multilevel Regulation of beta-Catenin Activity by SETD2 Suppresses the Transition from Polycystic Ki..
来源:CANCER RES( P 0008-5472 E 1538-7445 ) 发表时间: 2021/07
类型:期刊论文 为本人加分:14.183132
贡献度:参与作者
10 IP3 receptors regulate vascular smooth muscle contractility and hypertension
来源:JCI INSIGHT( P 2379-3708 E ) 发表时间: 2016/10
类型:期刊论文 为本人加分:12.850080