Mec1-independent activation of the Rad53 checkpoint kinase revealed by quantitative analysis of protein localization dynamics
Author:
Affiliation:
1. Department of Biochemistry, University of Toronto
2. Donnelly Centre for Cellular and Biomolecular Research, University of Toronto
3. The Weill Institute for Cell & Molecular Biology, Cornell University
Abstract
Funder
Canadian Institutes of Health Research
Natural Sciences and Engineering Research Council of Canada
National Institutes of Health
Publisher
eLife Sciences Publications, Ltd
Subject
General Immunology and Microbiology,General Biochemistry, Genetics and Molecular Biology,General Medicine,General Neuroscience
Link
https://cdn.elifesciences.org/articles/82483/elife-82483-v1.pdf
Reference90 articles.
1. Causes of genome instability;Aguilera;Annual Review of Genetics,2013
2. Mrc1 Transduces signals of DNA replication stress to activate Rad53;Alcasabas;Nature Cell Biology,2001
3. Synthetic genetic array (SGA) analysis in Saccharomyces cerevisiae and Schizosaccharomyces pombe;Baryshnikova;Methods in Enzymology,2010
4. Direct kinase-to-kinase signaling mediated by the FHA Phosphoprotein recognition domain of the Dun1 DNA damage Checkpoint kinase;Bashkirov;Molecular and Cellular Biology,2003
5. Phosphoproteomics reveals distinct modes of Mec1/ATR signaling during DNA replication;Bastos de Oliveira;Molecular Cell,2015
Cited by 2 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. Rad53 regulates the lifetime of Rdh54 at homologous recombination intermediates;Nucleic Acids Research;2023-10-18
2. Rad53 regulates the lifetime of Rdh54 at homologous recombination intermediates;2023-05-15
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