The Alzheimer’s disease–linked protease BACE2 cleaves VEGFR3 and modulates its signaling-Reference-Cited by-同舟云学术

The Alzheimer’s disease–linked protease BACE2 cleaves VEGFR3 and modulates its signaling

Published:2024-06-18 Issue:16 Volume:134 Page:
ISSN:1558-8238
Container-title:Journal of Clinical Investigation
language:en
Short-container-title:

Author:

Schmidt Andree,Hrupka Brian,van Bebber Frauke,Sunil Kumar Sanjay,Feng Xiao,Tschirner Sarah K.,Aßfalg Marlene,Müller Stephan A.,Hilger Laura Sophie,Hofmann Laura I.,Pigoni Martina,Jocher Georg^ORCID,Voytyuk Iryna^ORCID,Self Emily L.,Ito Mana,Hyakkoku Kana,Yoshimura Akimasa,Horiguchi Naotaka,Feederle Regina,De Strooper Bart,Schulte-Merker Stefan,Lammert Eckhard,Moechars Dieder,Schmid Bettina,Lichtenthaler Stefan F.^ORCID

Funder

Deutsche Forschungsgemeinschaft

Munich Cluster for Systems Neurology

Federal Ministry of Education and Research

Helmholtz Association

Institute for the Promotion of Innovation by Science and Technology

Alzheimer’s Association

Publisher

American Society for Clinical Investigation

Link

https://www.jci.org/articles/view/170550/files/pdf

Reference73 articles.

1. The β-Secretase BACE1 in Alzheimer’s Disease

2. The case for low-level BACE1 inhibition for the prevention of Alzheimer disease

3. Randomized Trial of Verubecestat for Mild-to-Moderate Alzheimer’s Disease

4. Randomized Trial of Verubecestat for Prodromal Alzheimer’s Disease

5. Efficacy and Safety of Lanabecestat for Treatment of Early and Mild Alzheimer Disease

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