Imidacloprid, Thiacloprid, and Their Imine Derivatives Up-Regulate the α4β2 Nicotinic Acetylcholine Receptor in M10 Cells

Author:

Tomizawa Motohiro,Casida John E.

Publisher

Elsevier BV

Subject

Pharmacology,Toxicology

Reference28 articles.

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2. Mechanism of up-regulation of neuronal nicotinic acetylcholine receptors in clonal cell lines and primary cultures of fetal rat brain;Bencherif;J. Pharmacol. Exp. Ther.,1995

3. Evidence that tobacco smoking increases the density of (-)-[3H]nicotine binding sites in human brain;Benwell;J. Neurochem.,1988

4. Upregulation of surface α4β2 nicotinic receptors is initiated by receptor desensitization after chronic exposure to nicotine;Fenster;J. Neurosci.,1999

5. A subtype of nicotinic cholinergic receptor in rat brain is composed of α4 and β2 subunits and is up-regulated by chronic nicotine treatment;Flores;Mol. Pharmacol.,1992

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