Overproduction of Cu/Zn-Superoxide Dismutase or Bcl-2 Prevents the Brain Mitochondrial Respiratory Dysfunction Induced by Glutathione Depletion-Reference-Cited by-同舟云学术

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Overproduction of Cu/Zn-Superoxide Dismutase or Bcl-2 Prevents the Brain Mitochondrial Respiratory Dysfunction Induced by Glutathione Depletion

Published:1999-08 Issue:2 Volume:158 Page:428-436
ISSN:0014-4886
Container-title:Experimental Neurology
language:en
Short-container-title:Experimental Neurology

Author:

Mérad-Saı̈doune Mansouria,Boitier Eric,Nicole Annie,Marsac Cécile,Martinou Jean Claude,Sola Brigitte,Sinet Pierre-Marie,Ceballos-Picot Irène

Publisher

Elsevier BV

Subject

Developmental Neuroscience,Neurology

Reference67 articles.

1. Determination of glutathione and glutathione dissulfide in biological samples;Anderson;Methods Enzymol.,1985

2. Apoptosis and autophagy in nigral neurons of patients with Parkinson's disease;Anglade;Histol. Histopathol.,1997

3. 3-Nitropropionic acid neurotoxicity is attenuated in Cu/Zn-superoxide dismutase transgenic mice;Beal;J. Neurochem.,1995

4. Assessment of the role of the glutathione and pentose phosphate pathways in the protection of primary cerebrocortical cultures from oxidative stress;Ben-Yoseph;J. Neurochem.,1996

5. Sequential damage in mitochondrial complexes by peroxidative stress;Benzi;Neurochem. Res.,1991

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