Homologous Recombination and Replication Fork Protection: BRCA2 and More!
Author:
Publisher
Cold Spring Harbor Laboratory
Subject
Genetics,Molecular Biology,Biochemistry
Reference92 articles.
1. ES cells do not activate p53-dependent stress responses and undergo p53-independent apoptosis in response to DNA damage
2. Average Risks of Breast and Ovarian Cancer Associated with BRCA1 or BRCA2 Mutations Detected in Case Series Unselected for Family History: A Combined Analysis of 22 Studies
3. BRCA2 acts as a RAD51 loader to facilitate telomere replication and capping
4. Replication stress: getting back on track
5. SMARCAL1 catalyzes fork regression and Holliday junction migration to maintain genome stability during DNA replication
Cited by 53 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. CaMKK2 and CHK1 phosphorylate human STN1 in response to replication stress to protect stalled forks from aberrant resection;Nature Communications;2023-11-30
2. Structural basis for stabilisation of the RAD51 nucleoprotein filament by BRCA2;Nature Communications;2023-11-02
3. Human HELQ regulates DNA end resection at DNA double-strand breaks and stalled replication forks;Nucleic Acids Research;2023-10-28
4. RAD51 paralogs synergize with RAD51 to protect reversed forks from cellular nucleases;Nucleic Acids Research;2023-10-16
5. Break‐induced RNA–DNA hybrids (BIRDHs) in homologous recombination: friend or foe?;EMBO reports;2023-10-11
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