Genetic background conversion ameliorates semi-lethality and permits behavioral analyses in cystathionine β-synthase-deficient mice, an animal model for hyperhomocysteinemia

Author:

Akahoshi Noriyuki,Kobayashi Chiho,Ishizaki Yasuki,Izumi Takashi,Himi Toshiyuki,Suematsu Makoto,Ishii Isao

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Reference57 articles.

1. Homocysteine, B vitamins, and risk of cardiovascular disease;Robinson;Heart,2000

2. Disorders of transsulfuration;Mudd,2001

3. Hyperhomocysteinemia: a new risk factor for degenerative diseases;Herrmann;Clin. Lab.,2002

4. Endothelial dysfunction and elevation of S-adenosylhomocysteine in cystathionine β-synthase-deficient mice;Dayal;Circ. Res.,2001

5. Role of hyperhomocysteinemia in endothelial dysfunction and atherothrombotic disease;Austin;Cell Death Differ.,2004

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