β-Amyloid Induces Pathology-Related Patterns of Tau Hyperphosphorylation at Synaptic Terminals

Author:

Wu Hsin-Yi1,Kuo Po-Cheng2,Wang Yi-Ting3,Lin Hao-Tai2,Roe Allyson D4,Wang Bo Y2,Han Chia-Li5,Hyman Bradley T4,Chen Yu-Ju23,Tai Hwan-Ching2

Affiliation:

1. Instrumentation Center, National Taiwan University, Taipei, Taiwan

2. Department of Chemistry, National Taiwan University, Taipei, Taiwan

3. Institute of Chemistry, Academia Sinica, Taipei, Taiwan

4. Department of Neurology, Massachusetts General Hospital, Harvard Medical School, Boston, Massachusetts

5. Master Program for Clinical Pharmacogenomics and Pharmacoproteomics, College of Pharmacy, Taipei Medical University, Taipei, Taiwan

Funder

Ministry of Science and Technology

Publisher

Oxford University Press (OUP)

Subject

Cellular and Molecular Neuroscience,Clinical Neurology,Neurology,General Medicine,Pathology and Forensic Medicine

Reference94 articles.

1. Neuropathology of Alzheimer's disease: What is new since A. Alzheimer?;Braak;Eur Arch Psychiatry Clin Neurosci,1999

2. Neuropathological alterations in Alzheimer disease;Serrano-Pozo;Cold Spring Harb Perspect Med,2011

3. Amyloid-β and tau—A toxic pas de deux in Alzheimer's disease;Ittner;Nat Rev Neurosci,2011

4. The intersection of amyloid beta and tau at synapses in Alzheimer's disease;Spires-Jones;Neuron,2014

5. The genetics of Alzheimer disease;Tanzi;Cold Spring Harb Perspect Med,2012

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