Muscle Protein Breakdown and the Critical Role of the Ubiquitin-Proteasome Pathway in Normal and Disease States
Author:
Affiliation:
1. Department of Cell Biology, Harvard Medical School, Boston, Massachusetts 02115
2. Renal Division, Emory University School of Medicine, Atlanta, Georgia 30322
Publisher
Oxford University Press (OUP)
Subject
Nutrition and Dietetics,Medicine (miscellaneous)
Link
http://academic.oup.com/jn/article-pdf/129/1/227S/24044398/4w01x90s227.pdf
Reference105 articles.
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3. In vivo half-life of a protein is a function of its amino-terminal residue.;Bachmair;Science,1986
4. The acidosis of chronic renal failure activates muscle proteolysis in rats by augmenting transcription of genes encoding proteins of the ATP-dependent ubiquitin-proteasome pathway.;Bailey;J. Clin. Invest.,1996
5. Activation of the ATP-ubiquitin-proteasome pathway in skeletal muscle of cachectic rats bearing a hepatoma.;Baracos;Am. J. Physiol.,1995
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