Sarcolipin deletion in mdx mice impairs calcineurin signalling and worsens dystrophic pathology-Reference-Cited by-同舟云学术

Sarcolipin deletion in mdx mice impairs calcineurin signalling and worsens dystrophic pathology

Published:2018-09-04 Issue: Volume: Page:
ISSN:0964-6906
Container-title:Human Molecular Genetics
language:en
Short-container-title:

Author:

Fajardo Val A¹,Chambers Paige J¹,Juracic Emma S¹,Rietze Bradley A¹,Gamu Daniel¹,Bellissimo Catherine¹,Kwon Frenk¹,Quadrilatero Joe¹,Russell Tupling A¹

Affiliation:

1. Department of Kinesiology, University of Waterloo, Waterloo, ON N2L 3G1 Canada

Funder

Canadian Institutes of Health Research

Publisher

Oxford University Press (OUP)

Subject

Genetics (clinical),Genetics,Molecular Biology,General Medicine

Link

http://academic.oup.com/hmg/advance-article-pdf/doi/10.1093/hmg/ddy302/25682203/ddy302.pdf

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3. Calcineurin-NFAT signaling, together with GABP and peroxisome PGC-1{alpha}, drives utrophin gene expression at the neuromuscular junction;Angus;Am. J. Physiol. Cell Physiol.,2005

4. Stimulation of calcineurin signaling attenuates the dystrophic pathology in mdx mice;Chakkalakal;Hum. Mol. Genet.,2004

5. The functional role of calcineurin in hypertrophy, regeneration, and disorders of skeletal muscle;Sakuma;J. Biomed. Biotechnol.,2010

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