βAPP Processing Drives Gradual Tau Pathology in an Age-Dependent Amyloid Rat Model of Alzheimer’s Disease-Reference-Cited by-同舟云学术

βAPP Processing Drives Gradual Tau Pathology in an Age-Dependent Amyloid Rat Model of Alzheimer’s Disease

Published:2017-10-18 Issue:11 Volume:28 Page:3976-3993
ISSN:1047-3211
Container-title:Cerebral Cortex
language:en
Short-container-title:

Author:

Audrain Mickael¹²³,Souchet Benoit¹³⁴,Alves Sandro¹³,Fol Romain¹²³,Viode Arthur⁵,Haddjeri Alexis⁶,Tada Satoru¹³,Orefice Nicola S¹³,Joséphine Charlène³⁷,Bemelmans Alexis-Pierre³⁷,Delzescaux Thierry³⁷,Déglon Nicole⁸⁹,Hantraye Philippe³⁷¹⁰,Akwa Yvette¹¹,Becher François⁵,Billard Jean-Marie⁶,Potier Brigitte⁶,Dutar Patrick⁶,Cartier Nathalie¹³,Braudeau Jérôme¹³^ORCID

Affiliation:

1. INSERM UMR1169, Université Paris-Sud, Université Paris-Saclay, Orsay, France

2. Université Paris Descartes, Paris, France

3. CEA, DRF, Institut Francois Jacob, MIRCen, Fontenay-aux-Roses, France

4. Université Paris Saclay, Paris, France

5. CEA, Institut Frédéric Joliot, Service de Pharmacologie et d’Immunoanalyse, Gif-sur-Yvette, France

6. INSERM UMR894, Centre de Psychiatrie et Neurosciences, Université Paris Descartes, Sorbonne Paris Cité, Paris, France

7. CNRS UMR9199, Fontenay-aux-Roses 92265, Université Paris-Sud, Université Paris-Saclay, Orsay, France

8. Department of Clinical Neurosciences, Laboratory of Cellular and Molecular Neurotherapies, Lausanne University Hospital, Lausanne, Switzerland

9. Neuroscience Research Center, Laboratory of Cellular and Molecular Neurotherapies, Lausanne University Hospital, Lausanne, Switzerland

10. INSERM UMS27, Fontenay-aux-Roses 92265, Université Paris-Sud, Université Paris-Saclay, Orsay, France

11. INSERM U1195 and Université Paris Sud and Université Paris-Saclay, 80 rue du général Leclerc, Le Kremlin-Bicêtre, France

Funder

DIM Biotherapies

the Région Ile-de-France

IDEX Paris Saclay

INSERM Transfert

CEA

Investissement d’Avenir

Publisher

Oxford University Press (OUP)

Subject

Cellular and Molecular Neuroscience,Cognitive Neuroscience

Link

http://academic.oup.com/cercor/article-pdf/28/11/3976/27082690/bhx260.pdf

Reference35 articles.

1. Laboratory animals: the Renaissance rat;Abbott;Nature,2004

2. Engineering theranostic nanovehicles capable of targeting cerebrovascular amyloid deposits;Agyare;J Control Release,2014

3. Ultramicroscopy as a novel tool to unravel the tropism of AAV gene therapy vectors in the brain;Alves;Sci Rep,2016

4. Alzheimer’s disease-like APP processing in wild-type mice identifies synaptic defects as initial steps of disease progression;Audrain;Mol Neurodegener,2016

5. Specific tau phosphorylation sites correlate with severity of neuronal cytopathology in Alzheimer’s disease;Augustinack;Acta Neuropathol,2002

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