Formation des dépôts d’IgA dans la maladie de Berger : ce que révèle le modèle animal
Author:
Publisher
EDP Sciences
Subject
General Biochemistry, Genetics and Molecular Biology
Link
http://www.biologie-journal.org/10.1051/jbio/2013022/pdf
Reference37 articles.
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2. Berger J., Yaneva H., Nabarra B., Barbanel C., Recurrence of mesangial deposition of IgA after renal transplantation.Kidney Int, 1975, 7, 232–241.
3. Berthelot L., Papista C., Maciel T.T., Biarnes-Pelicot M., Tissandie E., Wang P.H., Tamouza H., Jamin A., Bex-Coudrat J., Gestin A., Boumediene A., Arcos-Fajardo M., England P., Pillebout E., Walker F., Daugas E., Vrtosvnik F., Flamant M., Benhamou M., Cogné M., Moura I.C., Monteiro R.C.,Transglutaminase is essential for IgA nephropathy development acting through IgA receptors.J Exp Med, 2012, 209, 793–806.
4. Galla J.H., IgA nephropathy.Kidney Int, 1995, 47, 377–387.
5. Gharavi A.G., Moldoveanu Z., Wyatt R.J., Barker C.V., Woodford S.Y., Lifton R.P., Mestecky J., Novak J., Julian B.A., Aberrant IgA1 glycosylation is inherited in familial and sporadic IgA nephropathy.J Am Soc Nephrol, 2008, 19, 1008–1014.
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