Stand by me(mory): Chronic infection diminishes memory pool via IL-6/STAT1
Author:
Affiliation:
1. Department of Molecular Medicine II, Medical Faculty, Heinrich Heine University, Düsseldorf, Germany
2. Institute of Immunology, Medical Faculty, University of Duisburg-Essen, Essen, Germany
Abstract
Publisher
Rockefeller University Press
Subject
Immunology,Immunology and Allergy
Link
http://rupress.org/jem/article-pdf/216/3/474/1171108/jem_20190066.pdf
Reference15 articles.
1. Chronic virus infection compromises memory bystander T cell function in an IL-6/STAT1-dependent manner
2. Natural killer cells regulate diverse T cell responses
3. An Interleukin-21- Interleukin-10-STAT3 Pathway Is Critical for Functional Maturation of Memory CD8+ T Cells
4. Late Interleukin-6 Escalates T Follicular Helper Cell Responses and Controls a Chronic Viral Infection
5. Signal transducer and activator of transcription 3 (STAT3) mutations underlying autosomal dominant hyper-IgE syndrome impair human CD8+ T-cell memory formation and function
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