Poly(ADP-Ribose) Polymerase in the Cellular Response to DNA Damage, Apoptosis, and Disease
Author:
Publisher
Elsevier BV
Subject
Genetics (clinical),Genetics
Reference49 articles.
1. Defective induction but normal activation and function of p53 in mouse cells lacking poly-ADP-ribose polymerase;Agarwal;Oncogene,1997
2. Tumor necrosis factor-alpha: a mediator of focal ischemic brain injury;Barone;Stroke,1997
3. Mice lacking the poly(ADP-ribose) polymerase gene are resistant to pancreatic beta-cell destruction and diabetes development induced by streptozocin;Burkart;Nat Med,1999
4. Poly(ADP-ribose) polymerase binds with transcription enhancer factor 1 to MCAT1 elements to regulate muscle-specific transcription;Butler;Mol Cell Biol,1999
5. XRCC1 polypeptide interacts with DNA polymerase β and possibly poly(ADP-ribose) polymerase, and DNA ligase III is a novel molecular “nick sensor” in vitro;Caldecott;Nucleic Acids Res,1996
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