Genetic deficiency and pharmacological modulation of RORα regulate laser-induced choroidal neovascularization-Reference-Cited by-同舟云学术

Genetic deficiency and pharmacological modulation of RORα regulate laser-induced choroidal neovascularization

Published:2023-01-10 Issue:1 Volume:15 Page:37-52
ISSN:1945-4589
Container-title:Aging
language:en
Short-container-title:Aging

Author:

Liu Chi-Hsiu¹,Yemanyi Felix¹,Bora Kiran¹,Kushwah Neetu¹,Blomfield Alexandra K.¹,Kamenecka Theodore M.²,SanGiovanni John Paul³,Sun Ye¹,Solt Laura A.²⁴,Chen Jing¹

Affiliation:

1. Department of Ophthalmology, Boston Children’s Hospital, Harvard Medical School, Boston, MA 02115, USA

2. Department of Molecular Medicine, UF Scripps Biomedical Research, Jupiter, FL 33458, USA

3. BIO5 Institute and Department of Nutritional Sciences, University of Arizona, Tucson, AZ 85719, USA

4. Department of Immunology and Microbiology, UF Scripps Biomedical Research, Jupiter, FL 33458, USA

Publisher

Impact Journals, LLC

Subject

Cell Biology,Aging

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3. Experimental subretinal neovascularization is inhibited by adenovirus-mediated soluble VEGF/flt-1 receptor gene transfection: a role of VEGF and possible treatment for SRN in age-related macular degeneration.;Ueno;Gene Ther,2000

4. Prevention of experimental choroidal neovascularization with intravitreal anti-vascular endothelial growth factor antibody fragment.;Miller;Arch Ophthalmol,2002

5. VEGF is major stimulator in model of choroidal neovascularization.;Campochiaro;Invest Ophthalmol Vis Sci,2000

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