β‐Arrestin1 enhances liver fibrosis through autophagy‐mediated Snail signaling-Reference-Cited by-全球学者库

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β‐Arrestin1 enhances liver fibrosis through autophagy‐mediated Snail signaling

Published:2018-09-14 Issue:2 Volume:33 Page:2000-2016
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Tan Siwei¹²,Lu Yu¹²,Xu Minyi¹,Huang Xiaoli¹,Liu Huiling¹,Jiang Jie¹,Wu Bin¹²

Affiliation:

1. Department of GastroenterologyThe Third Affiliated Hospital of Sun Yat-Sen UniversityGuangzhouChina

2. Guangdong Provincial Key Laboratory of Liver Disease ResearchGuangzhouChina

Funder

National Natural Science Foundation of China

Sun Yat-sen University

Young Teacher Foundation of Sun Yat-sen University

Scientific Research and Technology Development Program of Guangxi

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.201800828RR

Reference50 articles.

1. Autophagy in Hepatic Fibrosis

2. Hepatocyte nuclear factor 4α attenuates hepatic fibrosis in rats

3. Pathobiology of liver fibrosis: a translational success story

4. Hepatic stellate cells in liver development, regeneration, and cancer

5. An HNF1α-regulated feedback circuit modulates hepatic fibrogenesis via the crosstalk between hepatocytes and hepatic stellate cells

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