Regulation of capacitative calcium entries by α1‐syntrophin: association of TRPC1 with dystrophin complex and the PDZ domain of α1‐syntrophin
Author:
Affiliation:
1. Institut de Physiologie et Biologie Cellulaire CNRS, UMR‐6187 University of Poitiers Poitiers France
2. LGCM CHU de Poitiers Poitiers France
Funder
College of Natural Resources and Sciences, Humboldt State University
Publisher
Wiley
Subject
Genetics,Molecular Biology,Biochemistry,Biotechnology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.06-6683com
Reference40 articles.
1. Calcium Influx through Calcium Leak Channels Is Responsible for the Elevated Levels of Calcium-dependent Proteolysis in Dystrophic Myotubes
2. Elevated subsarcolemmal Ca2+ in mdx mouse skeletal muscle fibers detected with Ca2+-activated K+ channels
3. Increased calcium influx in dystrophic muscle.
4. Increased calcium entry into dystrophin‐deficient muscle fibres of MDX and ADR‐MDX mice is reduced by ion channel blockers
5. Abnormal calcium homeostasis in Duchenne muscular dystrophy myotubes contracting in vitro
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