In vivo targeting of SF/HGF and c‐met expression via U1snRNA/ribozymes inhibits glioma growth and angiogenesis and promotes apoptosis-Reference-Cited by-同舟云学术

In vivo targeting of SF/HGF and c‐met expression via U1snRNA/ribozymes inhibits glioma growth and angiogenesis and promotes apoptosis

Published:2001-11-29 Issue:1 Volume:16 Page:1-16
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Abounader Roger¹²,Lal Bachchu²,Luddy Carey²,Koe Gary³,Davidson Beverly⁴,Rosen Eliot M.⁵,Laterra John¹⁶⁷²

Affiliation:

1. Johns Hopkins University School of MedicineDepartment of NeurologyBaltimoreMD

2. Johns Hopkins University School of MedicineKennedy Krieger Research InstituteBaltimoreMD

3. Valentis, Inc.BurlingameCA

4. University of Iowa College of MedicineIowa CityIA

5. Long Island Jewish Medical CenterAlbert Einstein College of MedicineNew Hyde ParkNY

6. Johns Hopkins University School of MedicineDepartment of OncologyBaltimoreMD

7. Johns Hopkins University School of MedicineDepartment of NeuroscienceBaltimoreMD

Funder

National Institutes of Health

Elsa U. Pardee Foundation

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.01-0421fje

Reference33 articles.

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2. Immunoreactive hepatocyte growth factor is a strong and independent predictor of recurrence and survival in human breast cancer;Yamashita J.;Cancer Res.,1994

3. Met and hepatocyte growth factor/scatter factor expression in human gliomas;Koochekpour S.;Cancer Res.,1997

4. Scatter factor promotes motility of human glioma and neuromicrovascular endothelial cells

5. Regulation of uveal melanoma interconverted phenotype by hepatocyte growth factor/scatter factor (HGF/SF);Hendrix M. J.;Am. J. Pathol.,1998

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