Dual modulation of both lipid oxidation and synthesis by peroxisome proliferator‐activated receptor‐γ coactivator‐1α and ‐1β in cultured myotubes
Author:
Affiliation:
1. Research DivisionJoslin Diabetes CenterBoston Massachusetts USA
2. Harvard Medical SchoolBoston Massachusetts USA
3. University of California–Los Angeles School of MedicineLos Angeles CA USA
4. SIDMAP, LLCLos Angeles California USA
Funder
National Institute of Diabetes and Digestive and Kidney Diseases
Boston Area Diabetes Endocrinology Research Center
Publisher
Wiley
Subject
Genetics,Molecular Biology,Biochemistry,Biotechnology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.09-133728
Reference57 articles.
1. The Coactivator PGC-1 Cooperates with Peroxisome Proliferator-Activated Receptor α in Transcriptional Control of Nuclear Genes Encoding Mitochondrial Fatty Acid Oxidation Enzymes
2. Regulation of hepatic fasting response by PPAR coactivator-1 (PGC-1): Requirement for hepatocyte nuclear factor 4 in gluconeogenesis
3. Transcriptional activators and coactivators in the nuclear control of mitochondrial function in mammalian cells
4. Err and Gabpa/b specify PGC-1 -dependent oxidative phosphorylation gene expression that is altered in diabetic muscle
5. Hyperlipidemic Effects of Dietary Saturated Fats Mediated through PGC-1β Coactivation of SREBP
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