Heart infarct in NOD‐SCID mice: Therapeutic vasculogenesis by transplantation of human CD34 + cells and low dose CD34 + KDR + cells-Reference-Cited by-同舟云学术

Heart infarct in NOD‐SCID mice: Therapeutic vasculogenesis by transplantation of human CD34 + cells and low dose CD34 + KDR + cells

Published:2004-07 Issue:12 Volume:18 Page:1392-1394
ISSN:0892-6638
Container-title:The FASEB Journal
language:en
Short-container-title:FASEB j.

Author:

Botta Rosanna¹²,Gao Erhe³,Stassi Giorgio⁴,Bonci Desirée²,Pelosi Elvira²,Zwas Donna¹,Patti Mariella⁴,Colonna Lucrezia⁵,Baiocchi Marta²,Coppola Simona²,Ma Xin³,Condorelli Gianluigi¹⁶⁷,Peschle Cesare¹²

Affiliation:

1. Kimmel Cancer Center, Thomas Jefferson UniversityPhiladelphiaPennsylvania19107

2. Department of Hematology, Oncology and Molecular MedicineIstituto Superiore Sanità00161RomeItaly

3. Center for Translational MedicineDepartment of MedicineThomas Jefferson UniversityPhiladelphiaPennsylvania19107

4. Department of Surgical and Oncological SciencesUniversity of PalermoItaly

5. Department of Medicine, Division of CardiologyThomas Jefferson UniversityPhiladelphiaPennsylvania19107

6. Laboratory of Molecular CardiologySan Raffaele Science Park of Rome00128Italy

7. Institute of Molecolar Medicine, University of CaliforniaSan Diego92093La JollaCalifornia

Publisher

Wiley

Subject

Genetics,Molecular Biology,Biochemistry,Biotechnology

Link

https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.03-0879fje

Reference26 articles.

1. Angiogenesis and vasculogenesis as therapeutic strategies for postnatal neovascularization

2. Transplantation of ex vivo expanded endothelial progenitor cells for therapeutic neovascularization

3. Implantation of Bone Marrow Mononuclear Cells Into Ischemic Myocardium Enhances Collateral Perfusion and Regional Function via Side Supply of Angioblasts, Angiogenic Ligands, and Cytokines

4. Therapeutic Potential of Ex Vivo Expanded Endothelial Progenitor Cells for Myocardial Ischemia

5. Neovascularization of ischemic myocardium by human bone-marrow–derived angioblasts prevents cardiomyocyte apoptosis, reduces remodeling and improves cardiac function

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