The fetal origins of atherosclerosis: maternal hypercholesterolemia, and cholesterol‐lowering or antioxidant treatment during pregnancy influence in utero programming and postnatal susceptibility to atherogenesis
Author:
Affiliation:
1. Department of Medicine 0682University of California San Diego La Jolla California USA
2. Department of MedicineFederico II University of Naples 80131 Naples Italy
Funder
National Heart, Lung, and Blood Institute
Publisher
Wiley
Subject
Genetics,Molecular Biology,Biochemistry,Biotechnology
Link
https://onlinelibrary.wiley.com/doi/pdf/10.1096/fj.02-0226rev
Reference90 articles.
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3. Fatty streak formation occurs in human fetal aortas and is greatly enhanced by maternal hypercholesterolemia. Intimal accumulation of low density lipoprotein and its oxidation precede monocyte recruitment into early atherosclerotic lesions.
4. Intracranial Arteries of Human Fetuses Are More Resistant to Hypercholesterolemia-Induced Fatty Streak Formation Than Extracranial Arteries
5. Pathophysiological Events during Pregnancy Influence the Development of Atherosclerosis in Humans
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