Advanced Oxidation Protein Products Promote Lipotoxicity and Tubulointerstitial Fibrosis via CD36/β-Catenin Pathway in Diabetic Nephropathy-Reference-Cited by-同舟云学术

Advanced Oxidation Protein Products Promote Lipotoxicity and Tubulointerstitial Fibrosis via CD36/β-Catenin Pathway in Diabetic Nephropathy

Published:2019-09-01 Issue:7 Volume:31 Page:521-538
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Li Xiao¹,Zhang Ting²,Geng Jian³,Wu Zhuguo⁴,Xu Liting²,Liu Jixing²,Tian Jianwei²,Zhou Zhanmei²,Nie Jing²,Bai Xiaoyan²

Affiliation:

1. Department of Emergency, Nanfang Hospital, Southern Medical University, Guangzhou, People's Republic of China.

2. Division of Nephrology, State Key Laboratory of Organ Failure Research, National Clinical Research Center for Kidney Disease, Guangdong Provincial Institute of Nephrology, Nanfang Hospital, Southern Medical University, Guangzhou, People's Republic of China.

3. Department of Pathology, Nanfang Hospital, Southern Medical University, Guangzhou, People's Republic of China.

4. Department of Internal Medicine, the Second Clinical Medical College, Guangdong Medical University, Dongguan, Guangdong, People's Republic of China.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

https://www.liebertpub.com/doi/pdf/10.1089/ars.2018.7634

Reference36 articles.

1. Blocking Wnt5a signaling decreases CD36 expression and foam cell formation in atherosclerosis

2. Differential TLR2 downstream signaling regulates lipid metabolism and cytokine production triggered by Mycobacterium bovis BCG infection

3. Palmitate deranges erythropoietin production via transcription factor ATF4 activation of unfolded protein response

4. CDK5 promotes renal tubulointerstitial fibrosis in diabetic nephropathy via ERK1/2/PPARγ pathway

5. A new model of diabetic nephropathy in C57BL/6 mice challenged with advanced oxidation protein products

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