Redox Regulation of Calpains: Consequences on Vascular Function-Reference-Cited by-同舟云学术

Redox Regulation of Calpains: Consequences on Vascular Function

Published:2019-03 Issue:7 Volume:30 Page:1011-1026
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Randriamboavonjy Voahanginirina¹²,Kyselova Anastasia¹²,Fleming Ingrid¹²

Affiliation:

1. Institute for Vascular Signalling, Centre for Molecular Medicine, Goethe University, Frankfurt am Main, Germany.

2. German Center of Cardiovascular Research (DZHK), Partner Site Rhein-Main, Frankfurt am Main, Germany.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

https://www.liebertpub.com/doi/pdf/10.1089/ars.2018.7607

Reference165 articles.

1. Calpain-2 activates Akt via TGF-β1-mTORC2 pathway in pulmonary artery smooth muscle cells

2. Antioxidant Improves Smooth Muscle Sarco/Endoplasmic Reticulum Ca 2+ -ATPase Function and Lowers Tyrosine Nitration in Hypercholesterolemia and Improves Nitric Oxide–Induced Relaxation

3. S-Glutathiolation by peroxynitrite activates SERCA during arterial relaxation by nitric oxide

4. Association of Calpain (CAPN) 10 (UCSNP-43, rs3792267) gene polymorphism with elevated serum androgens in young women with the most severe phenotype of polycystic ovary syndrome (PCOS)

5. Possible Involvement of m-Calpain in Vascular Smooth Muscle Cell Proliferation

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