Cross-Talk Between Mitochondria and NADPH Oxidase: Effects of Mild Mitochondrial Dysfunction on Angiotensin II-Mediated Increase in Nox Isoform Expression and Activity in Vascular Smooth Muscle Cells-Reference-Cited by-同舟云学术

Cross-Talk Between Mitochondria and NADPH Oxidase: Effects of Mild Mitochondrial Dysfunction on Angiotensin II-Mediated Increase in Nox Isoform Expression and Activity in Vascular Smooth Muscle Cells

Published:2009-06 Issue:6 Volume:11 Page:1265-1278
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Wosniak João¹,Santos Célio X. C.¹,Kowaltowski Alicia J.²,Laurindo Francisco R. M.¹

Affiliation:

1. Vascular Biology Laboratory, Heart Institute (InCor), University of São Paulo School of Medicine, São Paulo, Brazil.

2. Departamento de Bioquimica, Instituto de Quimica da Universidade de São Paulo, São Paulo, Brazil.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2009.2392

Reference59 articles.

1. The effects of ethidium bromide induced loss of mitochondrial DNA on mitochondrial phenotype and ultrastructure in a human leukemia T-cell line (MOLT-4 cells)

2. Increased cell-to-cell variation in gene expression in ageing mouse heart

3. Mitochondrial Integrity and Function in Atherogenesis

4. D-loop mutations in mitochondrial DNA: link with mitochondrial DNA depletion?

5. Hormone and neurotransmitter receptors in an established vascular endothelial cell line

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