The Crosstalk Between Nrf2 and AMPK Signal Pathways Is Important for the Anti-Inflammatory Effect of Berberine in LPS-Stimulated Macrophages and Endotoxin-Shocked Mice-Reference-Cited by-全球学者库

The Crosstalk Between Nrf2 and AMPK Signal Pathways Is Important for the Anti-Inflammatory Effect of Berberine in LPS-Stimulated Macrophages and Endotoxin-Shocked Mice

Published:2014-02 Issue:4 Volume:20 Page:574-588
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Mo Chunfen¹,Wang Ling¹,Zhang Jie¹,Numazawa Satoshi²,Tang Hong³,Tang Xiaoqiang¹,Han XiaoJuan¹,Li Junhong¹,Yang Ming¹,Wang Zhe¹,Wei Dandan¹,Xiao Hengyi¹

Affiliation:

1. Lab for Aging Research, Center for Medical Stem Cell Biology, State Key Laboratory of Biotherapy, West China Hospital, Sichuan University, Chengdu, China.

2. Department of Biochemical Toxicology, Showa University School of Pharmacy, Shinagawa, Tokyo, Japan.

3. Department of Infection Diseases, West China Hospital, Sichuan University, Chengdu, China.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2012.5116

Reference58 articles.

1. Induction of heme oxygenase 1 by moderately oxidized low-density lipoproteins in human vascular smooth muscle cells: Role of mitogen-activated protein kinases and Nrf2

2. LPS regulates proinflammatory gene expression in macrophages by altering histone deacetylase expression

3. Pre-Treatment of Recombinant Mouse MFG-E8 Downregulates LPS-Induced TNF-α Production in Macrophages via STAT3-Mediated SOCS3 Activation

4. Immunology in the Clinic Review Series; focus on metabolic diseases: development of islet autoimmune disease in type 2 diabetes patients: potential sequelae of chronic inflammation

5. AMPK inhibits fatty acid-induced increases in NF-κB transactivation in cultured human umbilical vein endothelial cells

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