Short Communication HTLV Type I Tax Activation of the CXCR4 Promoter by Association with Nuclear Respiratory Factor 1
Author:
Publisher
Mary Ann Liebert Inc
Subject
Infectious Diseases,Virology,Immunology
Link
http://www.liebertpub.com/doi/pdf/10.1089/088922299310728
Reference43 articles.
1. Pathogenesis of Diseases Induced by Human Lymphotropic Virus Type I Infection
2. HUMAN T CELL LEUKEMIA VIRUS TYPE I (HTLV-I) AND HUMAN DISEASES
3. Molecular mechanisms of human T-cell leukemia/lymphotropic virus type I infection
4. HTLV-1 oncoprotein Tax deregulates transcription of cellular genes through multiple mechanisms
5. Genetic characterization of transactivation of the human T-cell leukemia virus type 1 promoter: Binding of Tax to Tax-responsive element 1 is mediated by the cyclic AMP-responsive members of the CREB/ATF family of transcription factors
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2. T-2 toxin upregulates the expression of human cytochrome P450 1A1 (CYP1A1) by enhancing NRF1 and Sp1 interaction;Toxicology Letters;2019-10
3. The Human Cytomegalovirus US27 Gene Product Constitutively Activates Antioxidant Response Element-Mediated Transcription through G β γ, Phosphoinositide 3-Kinase, and Nuclear Respiratory Factor 1;Journal of Virology;2018-12
4. Tetramethylpyrazine in a Murine Alkali-Burn Model Blocks NFκB/NRF-1/CXCR4-Signaling-Induced Corneal Neovascularization;Investigative Opthalmology & Visual Science;2018-04-23
5. CXCR4: A virus’s best friend?;Infection, Genetics and Evolution;2014-07
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