p53, Aerobic Metabolism, and Cancer
Author:
Affiliation:
1. Center for Molecular Medicine, National Heart, Lung, and Blood Institute, National Institutes of Health, Bethesda, Maryland.
2. Department of Biomedical Laboratory Science, College of Health Science, Eulji University, GyeongGi-Do, Korea.
Publisher
Mary Ann Liebert Inc
Subject
Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry
Link
http://www.liebertpub.com/doi/pdf/10.1089/ars.2010.3650
Reference122 articles.
1. Role of p53 in Sensing Oxidative DNA Damage in Response to Reactive Oxygen Species-Generating Agents
2. Novel role of p53 in maintaining mitochondrial genetic stability through interaction with DNA Pol γ
3. Differential effects of spontaneous versus forced exercise in rats on the staining of parvalbumin-positive neurons in the hippocampal formation
4. NQO1 stabilizes p53 through a distinct pathway
5. Anticancer DNA intercalators cause p53-dependent mitochondrial DNA nucleoid re-modelling
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