Galectin-3, Galectin-9, and Interleukin-18 Are Associated with Monocyte/Macrophage Activation and Turnover More so than Simian Immunodeficiency Virus-Associated Cardiac Pathology or Encephalitis-Reference-Cited by-同舟云学术

Galectin-3, Galectin-9, and Interleukin-18 Are Associated with Monocyte/Macrophage Activation and Turnover More so than Simian Immunodeficiency Virus-Associated Cardiac Pathology or Encephalitis

Published:2024-09-01 Issue:9 Volume:40 Page:531-542
ISSN:0889-2229
Container-title:AIDS Research and Human Retroviruses
language:en
Short-container-title:AIDS Research and Human Retroviruses

Author:

Ding Andrew K.¹,Wallis Zoey K.¹,White Kevin S.¹,Sumer Cinar Efe¹,Kim Woong-Ki²³,Ardeshir Amir²³,Williams Kenneth C.¹^ORCID

Affiliation:

1. Department of Biology, Boston College, Chestnut Hill, Massachusetts, USA.

2. Division of Microbiology, Tulane National Primate Research Center, Tulane University, Covington, Louisiana, USA.

3. Department of Microbiology & Immunology, Tulane University School of Medicine, New Orleans, Louisiana, USA.

Publisher

Mary Ann Liebert Inc

Link

https://www.liebertpub.com/doi/pdf/10.1089/aid.2024.0008

Reference62 articles.

1. Monocyte/macrophages and their role in HIV neuropathogenesis

2. Galectin-3: An open-ended story

3. Galectin-9 as a biomarker of disease severity

4. Interleukin-18 Regulates Both Th1 and Th2 Responses

5. Galectin-3, a Marker of Cardiac Fibrosis, Predicts Incident Heart Failure in the Community

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