Hyperoxic Reperfusion after Global Cerebral Ischemia Promotes Inflammation and Long-Term Hippocampal Neuronal Death-Reference-Cited by-同舟云学术

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Hyperoxic Reperfusion after Global Cerebral Ischemia Promotes Inflammation and Long-Term Hippocampal Neuronal Death

Published:2010-04 Issue:4 Volume:27 Page:753-762
ISSN:0897-7151
Container-title:Journal of Neurotrauma
language:en
Short-container-title:Journal of Neurotrauma

Author:

Hazelton Julie L.¹²,Balan Irina¹²,Elmer Greg I.³,Kristian Tibor¹²,Rosenthal Robert E.¹²⁴,Krause Gary⁵,Sanderson Thomas H.⁵,Fiskum Gary¹²

Affiliation:

1. Department of Anesthesiology, University of Maryland School of Medicine, Baltimore, Maryland.

2. Center for Shock, Trauma, and Anesthesiology Research, University of Maryland School of Medicine, Baltimore, Maryland.

3. Department of Psychiatry, Maryland Psychiatric Research Center, University of Maryland School of Medicine, Baltimore, Maryland.

4. Program in Trauma, Department of Emergency Medicine, University of Maryland School of Medicine, Baltimore, Maryland.

5. Department of Emergency Medicine, Wayne State University School of Medicine, Detroit, Michigan.

Publisher

Mary Ann Liebert Inc

Subject

Neurology (clinical)

Link

http://www.liebertpub.com/doi/pdf/10.1089/neu.2009.1186

Reference28 articles.

1. Oximetry-Guided Reoxygenation Improves Neurological Outcome After Experimental Cardiac Arrest

2. Intraischemic but Not Postischemic Brain Hypothermia Protects Chronically following Global Forebrain Ischemia in Rats

3. Oxygen sensitivity of mitochondrial redox status and evoked potential recovery early during reperfusion in post-ischemic rat brain

4. Immunosuppression after traumatic or ischemic CNS damage: It is neuroprotective and illuminates the role of microglial cells

5. Neuroprotection for Ischaemic Stroke

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