Mito-Apocynin Prevents Mitochondrial Dysfunction, Microglial Activation, Oxidative Damage, and Progressive Neurodegeneration in MitoPark Transgenic Mice-Reference-Cited by-同舟云学术

Mito-Apocynin Prevents Mitochondrial Dysfunction, Microglial Activation, Oxidative Damage, and Progressive Neurodegeneration in MitoPark Transgenic Mice

Published:2017-11-10 Issue:14 Volume:27 Page:1048-1066
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Langley Monica¹,Ghosh Anamitra¹,Charli Adhithiya¹,Sarkar Souvarish¹,Ay Muhammet¹,Luo Jie¹,Zielonka Jacek²,Brenza Timothy³,Bennett Brian⁴,Jin Huajun¹,Ghaisas Shivani¹,Schlichtmann Benjamin³,Kim Dongsuk¹,Anantharam Vellareddy¹,Kanthasamy Arthi¹,Narasimhan Balaji³,Kalyanaraman Balaraman²,Kanthasamy Anumantha G.¹

Affiliation:

1. Department of Biomedical Sciences, Iowa Center for Advanced Neurotoxicology, Iowa State University, Ames, Iowa.

2. Department of Biophysics, Medical College of Wisconsin, Milwaukee, Wisconsin.

3. Department of Chemical and Biological Engineering, Iowa State University, Ames, Iowa.

4. Department of Physics, Marquette University, Milwaukee, Wisconsin.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2016.6905

Reference75 articles.

1. Pharmacological inhibition of neuronal NADPH oxidase protects against 1-methyl-4-phenylpyridinium (MPP+)-induced oxidative stress and apoptosis in mesencephalic dopaminergic neuronal cells

2. Association study of two genetic variants in mitochondrial transcription factor A (TFAM) in Alzheimer's and Parkinson's disease

3. Parkinsonâ€™s disease: animal models and dopaminergic cell vulnerability

4. Chronic microglial activation and progressive dopaminergic neurotoxicity

5. Selection and interpretation of clinical pathology indicators of hepatic injury in preclinical studies

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