HbE/β-Thalassemia and Oxidative Stress: The Key to Pathophysiological Mechanisms and Novel Therapeutics-Reference-Cited by-同舟云学术

HbE/β-Thalassemia and Oxidative Stress: The Key to Pathophysiological Mechanisms and Novel Therapeutics

Published:2017-05-10 Issue:14 Volume:26 Page:794-813
ISSN:1523-0864
Container-title:Antioxidants & Redox Signaling
language:en
Short-container-title:Antioxidants & Redox Signaling

Author:

Hirsch Rhoda Elison¹²,Sibmooh Nathawut³,Fucharoen Suthat⁴,Friedman Joel M.⁵

Affiliation:

1. Department of Medicine (Hematology), Albert Einstein College of Medicine, Bronx, New York.

2. Department of Anatomy and Structural Biology, Albert Einstein College of Medicine, Bronx, New York.

3. Department of Pharmacology, Faculty of Science, Mahidol University, Bangkok, Thailand.

4. Thalassemia Research Center, Institute of Molecular Biosciences, Mahidol University, Nakornpathom, Thailand.

5. Department of Physiology and Biophysics, Albert Einstein College of Medicine, Bronx, New York.

Publisher

Mary Ann Liebert Inc

Subject

Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry,Cell Biology,Clinical Biochemistry,Molecular Biology,Physiology,Biochemistry

Link

http://www.liebertpub.com/doi/pdf/10.1089/ars.2016.6806

Reference216 articles.

1. Consequence of β16 and β112 Replacements on the Kinetics of Hemoglobin Assembly

2. Significance of β116 His (G18) at α1β1 Contact Sites for αβ Assembly and Autoxidation of Hemoglobin

3. Cardiac involvement in thalassemia intermedia: a multicenter study

4. Haptoglobin: the hemoglobin detoxifier in plasma

5. Methemoglobinemia and ascorbate deficiency in hemoglobin E β thalassemia: metabolic and clinical implications

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