Rho and Reactive Oxygen Species at Crossroads of Endothelial Permeability and Inflammation
Author:
Affiliation:
1. Division of Pulmonary and Critical Care Medicine, Department of Medicine, University of Maryland, Baltimore, Maryland.
2. Department of Anesthesiology, University of Maryland School of Medicine, Baltimore, Maryland.
Publisher
Mary Ann Liebert Inc
Link
https://www.liebertpub.com/doi/pdf/10.1089/ars.2019.7798
Reference165 articles.
1. Rho and rho kinase modulation of barrier properties: cultured endothelial cells and intact microvessels of rats and mice
2. Direct Activation of RhoA by Reactive Oxygen Species Requires a Redox-Sensitive Motif
3. Phosphorylation and Activation of Myosin by Rho-associated Kinase (Rho-kinase)
4. RhoA/Rho-Associated Kinase Pathway Selectively Regulates Thrombin-Induced Intercellular Adhesion Molecule-1 Expression in Endothelial Cells via Activation of IκB Kinase β and Phosphorylation of RelA/p65
5. Polyethylene glycol superoxide dismutase and catalase attenuate increased blood-brain barrier permeability after ischemia in piglets.
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