The Role of Interleukin 10 and 18 in Chronic Spontaneous Urticaria Pathogenesis in the Context of Angioedema Coexistence
Author:
Affiliation:
1. Department of Internal Medicine with Division of Allergology, Institute of Medical Sciences, University of Opole, Opole, Poland.
Publisher
Mary Ann Liebert Inc
Subject
Virology,Cell Biology,Immunology
Link
https://www.liebertpub.com/doi/pdf/10.1089/jir.2020.0256
Reference36 articles.
1. The complex alteration in the network of IL-17-type cytokines in patients with hereditary angioedema
2. Interleukin-10 induces apoptosis in developing mast cells and macrophages
3. Oral nickel exposure may induce Type I hypersensitivity reaction in nickel-sensitized subjects
4. Airway epithelium expresses interleukin-18
5. Different expression patterns of plasma Th1-, Th2-, Th17- and Th22-related cytokines correlate with serum autoreactivity and allergen sensitivity in chronic spontaneous urticaria
Cited by 3 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. In silico Identification of Immune Cell-Types and Pathways Involved in Chronic Spontaneous Urticaria;Frontiers in Medicine;2022-07-07
2. The Role of Crosstalk of Immune Cells in Pathogenesis of Chronic Spontaneous Urticaria;Frontiers in Immunology;2022-05-31
3. Analysis of the Transcriptional Activity of Immune Response Genes in the Blood of Patients with Acute Urticaria;Open Access Macedonian Journal of Medical Sciences;2022-03-09
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