Bile-pancreatic juice exclusion increases p38MAPK activation and TNF-α production in ligation-induced acute pancreatitis in rats
Author:
Publisher
Elsevier BV
Subject
Gastroenterology,Hepatology,Endocrinology, Diabetes and Metabolism
Reference22 articles.
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2. Evidence of a central role for p38MAPK induction of tumor necrosis factor-α in pancreatitis-associated pulmonary injury;Yang;Surgery,1999
3. The role of cytokines in the pathogenesis of acute pancreatitis;Norman;Am J Surg,1998
4. Etiopathogenesis of acute pancreatitis;Karne;Surg Clin North Am,1999
5. CCK-A receptor induction and p38MAPK and NF-κB activation in acute pancreatitis;Samuel;Pancreatology,2004
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3. Significantly different clinical features between hypertriglyceridemia and biliary acute pancreatitis: a retrospective study of 730 patients from a tertiary center;BMC Gastroenterology;2018-06-19
4. Effects of 1,3,4-thiadiazine compound with antidepressant properties in ligation model of acute pancreatitis;General physiology and biophysics;2018
5. Up-regulation of Tight-Junction Proteins by p38 Mitogen-Activated Protein Kinase/p53 Inhibition Leads to a Reduction of Injury to the Intestinal Mucosal Barrier in Severe Acute Pancreatitis;Pancreas;2016-09
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