Interleukin-6 Signaling Blockade Induces Regulatory Plasmablasts in Neuromyelitis Optica Spectrum Disorder-Reference-Cited by-同舟云学术

Interleukin-6 Signaling Blockade Induces Regulatory Plasmablasts in Neuromyelitis Optica Spectrum Disorder

Published:2024-07 Issue:4 Volume:11 Page:
ISSN:2332-7812
Container-title:Neurology Neuroimmunology & Neuroinflammation
language:en
Short-container-title:Neurol Neuroimmunol Neuroinflamm

Author:

Akatani Ritsu¹^ORCID,Chihara Norio¹^ORCID,Hara Atsushi¹,Tsuji Asato¹,Koto Shusuke¹,Kobayashi Kazuhiro¹,Toda Tatsushi¹^ORCID,Matsumoto Riki¹^ORCID

Affiliation:

1. From the Division of Neurology (R.A., N.C., A.H., A.T., S.K., R.M.); Division of Molecular Brain Science (K.K.), Kobe University Graduate School of Medicine; and Department of Neurology (T.T.), Graduate School of Medicine, The University of Tokyo, Japan.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Link

https://www.neurology.org/doi/pdfdirect/10.1212/NXI.0000000000200266

Reference50 articles.

1. A serum autoantibody marker of neuromyelitis optica: distinction from multiple sclerosis

2. IgG marker of optic-spinal multiple sclerosis binds to the aquaporin-4 water channel

3. Interleukin 6 signaling promotes anti-aquaporin 4 autoantibody production from plasmablasts in neuromyelitis optica

4. Plasmablasts as Migratory IgG-Producing Cells in the Pathogenesis of Neuromyelitis Optica

5. CNS Aquaporin-4-specific B cells connect with multiple B-cell compartments in neuromyelitis optica spectrum disorder

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Association Between Serum Interleukin-32 Level and Disease Status in Cases with Neuromyelitis Optica Spectrum Disorders;Journal of Inflammation Research;2024-08