Author:
Arawaka S.,Saito Y.,Murayama S.,Mori H.
Abstract
In familial PD, a mutation of the α-synuclein gene has been identified. α-Synuclein also was revealed in Lewy bodies in idiopathic PD. Lewy bodies in neurodegeneration with brain iron accumulation type 1(NBIA 1; Hallervorden-Spatz syndrome) were found to show immunostaining for α-synuclein/precursor of non-A beta component of Alzheimer's disease amyloid, indicating that α-synuclein is commonly associated with the formation of Lewy bodies in other sporadic and familial neurodegenerative diseases apart from PD.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Cited by
154 articles.
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