Endothelial Cell–Derived Von Willebrand Factor, But Not Platelet-Derived, Promotes Atherosclerosis in Apolipoprotein E

Endothelial Cell–Derived Von Willebrand Factor, But Not Platelet-Derived, Promotes Atherosclerosis in Apolipoprotein E–Deficient Mice

Published:2018-03 Issue:3 Volume:38 Page:520-528
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:Arterioscler Thromb Vasc Biol.

Author:

Doddapattar Prakash¹,Dhanesha Nirav¹,Chorawala Mehul R.¹,Tinsman Chandler¹,Jain Manish¹,Nayak Manasa K.¹,Staber Janice M.¹,Chauhan Anil K.¹

Affiliation:

1. From the Department of Internal Medicine (P.D., N.D., M.R.C., M.J., M.K.N., A.K.C.) and Stead Family Department of Pediatrics (C.T., J.M.S.), University of Iowa, Iowa City.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference46 articles.

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2. ADAMTS-13 rapidly cleaves newly secreted ultralarge von Willebrand factor multimers on the endothelial surface under flowing conditions

3. The high molecular weight form of endothelial cell von Willebrand factor is released by the regulated pathway

4. Biosynthesis of von Willebrand protein by human megakaryocytes.

5. von Willebrand factor–mediated platelet adhesion is critical for deep vein thrombosis in mouse models

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