Endothelin-1–Induced Arrhythmogenic Ca2+Signaling Is Abolished in Atrial Myocytes of Inositol-1,4,5-Trisphosphate(IP3)–Receptor Type 2–Deficient Mice
Author:
Affiliation:
1. From the Department of Medicine (H.L., F.S., J.C.), University of California San Diego, La Jolla, Calif; and the Department of Physiology (A.V.Z., L.A.B.), Loyola University of Chicago, Stritch School of Medicine, Maywood, Ill.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Cardiology and Cardiovascular Medicine,Physiology
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