Abandoning M1/M2 for a Network Model of Macrophage Function
Author:
Affiliation:
1. From the Center for Systems Biology, Massachusetts General Hospital, Harvard Medical School, Boston.
Publisher
Ovid Technologies (Wolters Kluwer Health)
Subject
Cardiology and Cardiovascular Medicine,Physiology
Reference40 articles.
1. Differential Contribution of Monocytes to Heart Macrophages in Steady-State and After Myocardial Infarction
2. An Abundant Tissue Macrophage Population in the Adult Murine Heart with a Distinct Alternatively-Activated Macrophage Profile
3. Ly-6C high Monocytes Depend on Nr4a1 to Balance Both Inflammatory and Reparative Phases in the Infarcted Myocardium
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