An Oral ApoJ Peptide Renders HDL Antiinflammatory in Mice and Monkeys and Dramatically Reduces Atherosclerosis in Apolipoprotein E

An Oral ApoJ Peptide Renders HDL Antiinflammatory in Mice and Monkeys and Dramatically Reduces Atherosclerosis in Apolipoprotein E–Null Mice

Published:2005-09 Issue:9 Volume:25 Page:1932-1937
ISSN:1079-5642
Container-title:Arteriosclerosis, Thrombosis, and Vascular Biology
language:en
Short-container-title:ATVB

Author:

Navab Mohamad¹,Anantharamaiah G.M.¹,Reddy Srinivasa T.¹,Van Lenten Brian J.¹,Wagner Alan C.¹,Hama Susan¹,Hough Greg¹,Bachini Eugene¹,Garber David W.¹,Mishra Vinod K.¹,Palgunachari Mayakonda N.¹,Fogelman Alan M.¹

Affiliation:

1. From the David Geffen School of Medicine at UCLA (M.N., S.T.R., B.J.V.L., A.C.W., S.H., G.H., E.B., A.M.F.), Los Angeles, Calif; and the Department of Medicine (G.M.A., D.W.G., V.K.M., M.N.P.), Atherosclerosis Research Unit, University of Alabama at Birmingham.

Publisher

Ovid Technologies (Wolters Kluwer Health)

Subject

Cardiology and Cardiovascular Medicine

Reference25 articles.

1. Effect of Recombinant ApoA-I Milano on Coronary Atherosclerosis in Patients With Acute Coronary Syndromes

2. High-Density Lipoproteins as an Emerging Therapeutic Target for Atherosclerosis

3. Human apolipoprotein A-I and A-I mimetic peptides: potential for atherosclerosis reversal

4. Influenza Infection Promotes Macrophage Traffic Into Arteries of Mice That Is Prevented by D-4F, an Apolipoprotein A-I Mimetic Peptide

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